2.4 Assessment Details
2.4.1 Concept Map and Guided Question Response
|Type of Collaboration:||Individual|
|Due:||Week 5, Monday 19th August 2019 at 12 midnight|
|Submission:||Refer to Section 2.5 of the LG - General Submission Requirements|
|Format:||Concept Map (250 words) and Guided question response (1250 words) Total 1500 words|
Unit Learning Outcomes: 1,2,3,4
|Length:||250 words plus 1250 words (1500 words in total)|
There is a word limit of 1,500 words. Use your computer to total the number of words used in your assignment. However, do not include the reference list at the end of your assignment in the word count. In-text citations will be included in the additional 10% word count. If you exceed the word limit by more than 10% the marker will stop marking at 1650 words.
Aim of assessment
The purpose of this assessment is to enable students to:
You are to develop a concept map for the topic systolic heart failure and answer three questions related to an acute exacerbation systolic heart failure. References are to include six (6) recent, relevant references including journal articles, textbook material and evidence-based resources.
Concept Map (250 words)
– Severe dyspnoea
– Respiratory rate of 24 breaths/minute
– SpO2 85%, on room air
– BP 170/95mmHg
– Pulse rate of 120 beats/minute
– Auscultation of lungs identifies bilateral basal crackles
Mrs Brown was connected to an ECG monitor that showed atrial fibrillation. A diagnosis of acute exacerbation of chronic systolic heart failure was made.
Question 1. (650 words) (Learning outcome 1 and 5)
Explain the pathogenesis causing the clinical manifestations with which Mrs Brown presented.
Question 2. (400words) (Learning outcomes 2, 4 and 5)
Discuss two high priority nursing strategies to manage Mrs Brown and provide evidence-based rationales for these strategies.
Question 3. (200 words) (Learning outcome 3 and 5)
Two of the drugs that were given to Mrs Brown were IV Furosemide (Lasix) and sublingual Glyceryl Trinitrate (GTN). a. Discuss the mechanism of action of these two drugs, and relate to the underlying pathogenesis of an acute exacerbation of chronic systolic heart failure.
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